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Unlike patient-specific neoantigens, which require individualized immunotherapeutic development 1,2,3 , commonly mutated oncoprotein-derived epitopes presented on class I major histocompatibility complex (MHC) molecules provide an opportunity to target cancer-specific antigens shared across patient populations 4,5

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(Driscoll and Teichman, 2001)

demonstrated that ferritin delivery to lysosomes requires NCOA4, and NCOA4-deficient cells fail to degrade ferritin, leading to a reduction in bioavailable intracellular iron (53)