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The latter notion is supported by studies demonstrating that deletion of SSADH upregulates the expression of mitochondrial proteins (Andersen et al

TB-500 exerts effects through actin regulation, cellular migration, and tissue remodeling dynamics

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Research has suggested that increased activity of the endogenous transsulfuration pathway could improve cancer cell survival in environments with diminished extracellular cysteine levels (22)

revealed that GPX4 overexpression prevents basophilic leukemia cells in mice from death caused by oxidative damage (Imai et al., 1996), thus, theoretically, GPX4 overexpression could make cells resistant to oxidative stress-induced ferroptosis

doi: 10.3390/ijms151017920