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11 , e1005153 (2015)

In more recent work, LL-37 was also found to be implicated in AD, where it may promote disease by triggering membrane translocation and the activation of the chloride channel CLIC1, ultimately inducing microglial hyperactivation [100]

Not only is this a waste of time, but it also makes us look like we are on a witch hunt

Among the most discussed peptides in wellness and regenerative medicine are BPC157, KPV, and argininerich peptides (ARG)

(2021) mentioned inositol stabilized arginine as a substance that enhances neurotransmission [19], Decroix et al

6 Limitation and Future perspective Although current research has revealed the critical role of ferroptosis in the pathological progression of AD, several limitations remain to be addressed in this field: (1) Existing evidence primarily relies on animal models or in vitro cellular experiments, failing to comprehensively reflect the spatiotemporal dynamics of iron metabolism dysregulation in the brains of human AD patients
