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Tetrandrine has been approved for the treatment of silicosis in China, but it still cannot be cured

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Studies have demonstrated that tau protein exhibits abnormal interactions with mitochondria, disrupting their cellular functions and contributing to neuronal death and loss [4, 5]

CBA/CaJ mice were purchased from the Jackson Laboratory (Bar Harbor, ME)

Various studies have demonstrated that targeting ferroptosis, including mechanisms involving epidermal growth factor receptor, PI3K, ACSL4, and Nrf2, promotes ferroptosis by activating different pathways to induce gene mutation [167-169], ROS accumulation, and reduction in GSH and GPX4 levels

Looking ahead, by integrating genomic, proteomic, and metabolomic data from patients, therapies can be customized based on specific molecular profiles, significantly increasing the efficacy and safety of regenerative treatments