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In innate immunity, Clostridium modulates macrophage polarization (shifting from pro-inflammatory M1 to anti-inflammatory M2), induces tolerogenic dendritic cell phenotypes to suppress excessive antigen presentation, regulates inflammatory body activity and neutrophil hyperactivation, and modulates ILC subsets to reinforce mucosal defense

doi: 10.1016/0098-2997(93)90002-U, PMID: 17 Vander HeidenMGDeBerardinisRJ

A., Cabello, G., Marco, J

Haas, G

Raftos JE, Whillier S, Kuchel PW

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