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Effects of p-Cresol on senescence, survival, inflammation, and odontoblast differentiation in canine dental pulp stem cells

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[31] and we have further optimised conditions to minimise the problems of auto-oxidation and degradation for the study of oxidative stress in biological tissues

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Although it was speculated that the protective effects of PPAR might depend on its role in facilitating fatty acid catabolism, a recent study showed that this protection is due in part to antioxidant function of the PPAR target gene mitochondrial uncoupling protein 2 (UCP2) [93]