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Skinnider MA

Given that kidney is the organ for drug excretion, receiving almost 25% of the cardiac output, the renal arterioles and glomerular capillaries are significantly vulnerable to NSAIDs [171]

Zhou X, Li Y, Wang W, Wang S, Hou J, Zhang A, et al

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Mitophagy-dependent macrophage reprogramming protects against kidney fibrosis

In the mechanism of dithiol Grxs, the reduction of PSSG and mixed disulfides begins with a nucleophilic attack of the N -terminal cysteine, but GSH is released
