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[DOI] [PMC free article] [PubMed] [Google Scholar] 54.Lee B., II, Suh Y.S., Chung Y.J., Yu K., Park C.B

Feed-forward loops involving TGF- often contribute to fibrosis pathogenesis by exaggerating normal physiologic responses, driving their chronic persistence, and triggering inflammation (Figure 4A)

, Worringer, K

This pattern may reflect the peptides ability to address ongoing inflammation quickly while structural remodeling requires extended time regardless of treatment

Other nuclear factor binding sites, such as the PU.1 and GATA sites, may modulate expression during cell differentiation

doi: 10.1002/jcb.27371 111 ZhangX.WuJ.YeB.WangQ.XieX.ShenH