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These findings were not attributed to variations in food intake, which implies that the observed results were mediated by alterations in metabolic processes [1]

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This peroxidase activity is mediated by a reduced cysteine residue (also referred to as peroxidatic Cys) that donates its electron to neutralize peroxide and then recycle back into its native form with the help of reductants like thioredoxin (Prdx1-5) and glutathione (Prdx6)

Gmez-Lechn MJ, Cj V, Donato MT (2008) An update on metabolism studies using human hepatocytes in primary culture

It is likely that zonulin release is initiated by dysbiosis and gliadin by a similar mechanism

Classical homocystinuria: From cystathionine beta-synthase deficiency to novel enzyme therapies