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However, individual supplementation of FA did not show positive effects in this study

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Taking into consideration that mutants affected in glutathione biosynthesis exhibited a susceptible phenotype while, under our conditions, none of the tested sulfate transporter mutants did so, the latter did not seem to affect glutathione homeostasis sufficiently to result in increased susceptibility to AITC

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Conclusion: This study demonstrated that SQH ameliorated DbCM injury primarily by inhibiting ACSL4/ALOX15-mediated ferroptosis

Abnormal LM reduces glutathione levels, increasing cell susceptibility to oxidative damage