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In fact, reports of bacterial endotoxin in the pelvic cavity and menstrual blood of people with endometriosis 37 have led to a bacterial contamination hypothesis suggesting endotoxin/bacteria in the menstrual effluent contributes to pelvic inflammation, growth, and progression of endometriotic lesions (reviewed in 38,39 )

K., O'Connor T., Aloi J

This circumstance has made it difficult to translate mouse results to the clinic

Moreover, immunofluorescence results showed that Cdc42 and PKCz were colocalized on the cell membrane and the colocalization tended to be on one side rather than the uniform distribution throughout the cell membrane after treatment (Fig

Bibcode:2020NatCo..11.4276T

How these medications overlap, interact, and persist in your system affects both safety and efficacy