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Unlike conventional DAT blockers such as methylphenidate, the disruption of D2R-DAT leads to decreased membrane expression of DAT, while leaves the remaining DAT functional to facilitate the reuptake of dopamine [23]

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GHK-Cu is the copper(II) chelate of glycyl-L-histidyl-L-lysine, studied for fibroblast extracellular-matrix gene-expression modulation

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Type IIB focal cortical dysplasia with balloon cells in medial temporal lobe epilepsy: Clinical, neuroimaging, and histopathological findings