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It is produced by the liver and found in every cell of the body, playing a key role in protecting cells from oxidative damage, neutralizing free radicals, and aiding in detoxification

MR analyses leverage single nucleotide polymorphism (SNP) data from independent Genome-Wide Association Studies (GWAS) to conduct SNP-exposure and SNP-outcome association analyses, ultimately yielding causal effect estimates (27)

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Global TST gene silencing in mice displayed an apparently diabetogenic phenotype, the circulating sulfide showed a 13-fold and 10-fold elevation in plasma and blood, while thiosulfate, the main oxidative metabolite of H 2 S, showed a 20-fold and a 475-fold increase in plasma and urine compared to healthy wildtype controls [[39], [40], [41], [42]]