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Am J Clin Nutr 84 (5):1243

The mode of therapeutic action of NAC is believed to involve its hydrolysis in the affected cells to cysteine, leading to increased synthesis of GSH

10.7554/eLife.89319 130 MarriottI

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TGR inhibitors do not react covalently with GSH, selenocysteine, or TGR, and inhibition of TGR is reversible To evaluate stability of the TGR inhibitors in the presence of GSH and selenocysteine, compound 2 and a known covalent inhibitor of TGR, TRi-1 20,45 , were incubated with GSH or the N-Boc protected methyl ester of selenocysteine, and the reaction mixtures were analyzed by LCMS (Supplementary Figs