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Tumor-derived EVs can also enter into the circulation and prepare distant organs for metastasis by creating favorable conditions for tumor cell growth (128)

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When cysteinesulfinate itself was used as substrate, isolated hepatocytes, renal cortical tubules, enterocytes, perfused isolated hindquarter, and intact rats all exhibited a high capacity for cysteinesulfinate metabolism to carbon dioxide or sulfate, with rates of cysteinesulfinate oxidation far exceeding those for Cys catabolism to (Bagley and Stipanuk 1995

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(2000) Egr-1, a master switch coordinating upregulation of divergent gene families underlying ischemic stress