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The tail end of idebenone and MitoQ are further modified with a hydroxyl group or triphenylphosphonium moiety, respectively, with the latter exhibiting mitochondrial targeting

A., Chatfield, M

E2 deficiency during perimenopause is a major driver of mitochondrial bioenergetic impairment, with the ETC being a primary target

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These include the following: therapeutic benefits resulting from non-antioxidant mechanisms, insufficient dosage to elicit an antioxidant effect, poor oral bioavailability, a narrow therapeutic index, or toxicity that precludes clinical use