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Using them in a study is like trying to build a skyscraper on a foundation of sand
These changes disrupt the cell's ability to produce energy and repair itself
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This makes it a potential treatment option for even the worst kinds of back pain
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Similar to the human GCLC, up-regulation of the human GCLM by -NF involved binding of transcription factor Nrf2 (possibly in complexes with other Jun or Maf proteins as in GCLC) to a functional ARE/EpRE site located at 302 of the human GCLM [104,134]
