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In summary, GA inhibited the RhoA/CDC42 and PI3K-Akt-ERK1/2 pathways and activated the AMPK-eNOS-FAS signaling pathway, thereby suppressing the migration and proliferation of VSMCs (Figure 3)
The role of astrocytes in amyloid -protein toxicity and clearance
Genevestigator V3: a reference expression database for the meta-analysis of transcriptomes
Reduced proficiency in homologous recombination underlies the high sensitivity of embryonal carcinoma testicular germ cell tumors to Cisplatin and poly (adp-ribose) polymerase inhibition
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