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IFN- signaling plays a pivotal role in stimulating antitumor response mainly through activating cytolytic T cells and promoting tumor antigen presentation, 592 and IFN- signaling profile is related to response to ICBs

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However, knockdown or deletion of TET2 shifts the gene expression profiles of these myeloid cells toward a pro-inflammatory phenotype, impairing their suppressive capabilities and promoting anti-tumor immune responses [100]

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