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Notably, a deficiency in eNOS-derived NO is a primary contributor to endothelial dysfunction, while excessive iNOS-driven NO and nitrosative stress can cause cellular damage

Redox-responsive nanoparticles, triggered by glutathione, release drugs in ROS-rich environments, as shown by indomethacin-loaded polyprodrug amphiphiles reducing inflammation (6)

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25 (8), e515e529

Effects of estrogen with micronized progesterone on cortical and trabecular bone mass and microstructure in recently postmenopausal women

Tong Z, Li H, Goljer I, McConnell O, Chandrasekaran A
