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10.1016/j.pediatrneurol.2013.12.007 190 LiptonS

This has been validated with animal models, for example, PPAR knockout (KO) mice showed reduced FA and increased glucose oxidation, and were resistant to DCM development.28 Conversely, in hearts of diabetic mice that overexpress PPAR, higher myocardial triglyceride levels alongside a more severe DCM phenotype were observed.29 Fig