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Kroner et al (49) demonstrated that elevated iron levels in microglia promote phagocytosis and drive a harmful M1 phenotype, which triggers the release of pro-inflammatory factors such as TNF-, IL-1, IL-6 and nitric oxide (NO), causing neuronal damage

(2017) 16873-85:E11

By targeting these substrates, SIRT1 regulates processes such as telomere integrity, epigenetic regulation, intercellular communication, inflammation, and mitochondrial functionall of which are essential in mitigating cellular senescence in the liver (Mohammed et al., 2021)

Zou, Q.-Z

Selenium: This mineral is a key component of the enzymes that help glutathione neutralize free radicals

Astrocytes, from brain glue to communication elements: the revolution continues