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Contribution of methylenetetrahydrofolate reductase (MTHFR) polymorphisms to negative symptoms in schizophrenia

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In summation, while the potential of these selenium-enriched compounds is undeniably promising, there remains an imperative for further comprehensive investigation, particularly in vivo studies, to ascertain the consequences of their prolonged consumption and to evaluate the enduring impacts related to their sustained application

In Japanese acatalasemia II, a frame shift mutation occurs due to the deletion of thymine in position 358 of exon 4 which modifies the amino acid sequence and produces a new TGA (stop) codon at the 3 terminal

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