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doi: 10.1134/S0023158414020153

In VaD rats, QBT reversed aberrant changes associated with ferroptosis and pyroptosis by upregulating the nuclear factor erythroid 2-related factor 2 (Nrf2)/cystine-glutamate antiporter (xCT)/glutathione peroxidase 4 (GPX4) pathway, downregulating the nucleotide-binding oligomerization domain-like receptor family pyrin domain-containing 3 (NLRP3)/cysteine-requiring aspartate protease-1 (Caspase-1)/gasdermin D (GSDMD) pathway, and inhibiting both ferroptosis and pyroptosis in neuronal cells

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Nature 592 (7856), 695703

Lung cancer Tobacco smoke is a known risk factor for lung cancer development, and antioxidant enzymes, such as GPx-1, may play essential roles in eliminating pro-carcinogenic oxidants caused by smoke inhalation

The K 2P channels are open at (and hence contribute to) the resting membrane potential