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In addition, systemic treatment with the GSH precursor N-acetyl-cysteine (NAC) increased accumbal GSH levels and led to improved performance, potentially mediated by a cell-type specific shift in glutamatergic inputs to accumbal medium spiny neurons

NAC (N-Acetyl-L-Cysteine) supports glutathione levels indirectly by providing cysteine the rate limiting amino acid for glutathione synthesis and the body uses that cysteine to synthesize its own glutathione

Recent studies have provided compelling evidence supporting the notion that ferroptosis plays a key pathogenic role in many diseases such as various cancer types, neurodegenerative disease, diseases involving tissue and/or organ injury, and inflammatory and infectious diseases