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Activated mTOR signaling can trigger epithelialmesenchymal transition (EMT) and promote the maintenance of cancer stem cells (CSCs), resulting in chemoresistance in ovarian cancer patients, and treatment with BEZ235 (a dual PI3K/mTOR inhibitor) might be a promising approach for reversing chemoresistance [112]

On the other hand, oxidative stress leads to mtDNA damage, the damaged mtDNA will exacerbate ROS production, thus aggravating oxidative stress, and forming a vicious cycle

The one-carbon pathway also involves the methionine cycle, which itself functions to convert homocysteine into methionine

Normal levels of intracellular GSH may exert a critical negative control on the elaboration of proinflammatory cytokines

A-lipoic acid alleviates folic acid-induced renal damage through inhibition of ferroptosis

doi: 10.1093/jn/136.1.21