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Animal models reveal that metformin upregulated placental growth factor (PlGF) while suppressing soluble fms-like tyrosine kinase-1 (sFlt-1) production through AMPK-mediated pathways (Tong et al., 2022)
Here, to better define this clinically taxing and translationally challenging malignancy, we applied a multiomics approach, consisting of 13 modalities to analyze 173 treatment-naive individuals with AML
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