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Neuronal Survival: GHRP-6 is studied for its anti-apoptotic (anti-cell death) effects on neurons, particularly following an ischemic event (stroke)

One practical consideration for long-term research protocols: MK-677's continuous ghrelin receptor activation can increase fasting glucose and HbA1c levels in some study models, particularly those with pre-existing insulin resistance

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Cells were treated with either glutathione diethyl ester (GDE) to enhance intracellular GSH or L-buthionine sulfoximine (L-BSO) to inhibit GSH synthesis

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as a rule it is the loss of glial supportive or protective functions which damages neurones