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2003;123(3 Suppl):446S

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This is due to an impairment in dTMP synthesis which leads to cell cycle arrest in S-phase of rapidly proliferating cells, in particular hematopoietic cells

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IGF-1 LR3 promotes protein synthesis and muscle hypertrophy IGF-1 LR3 induces a high rate of cell proliferation (hyperplasia) and division due to remaining in the bloodstream for much longer than regular IGF-1, with the effect of increasing muscle cells

However, the C26S mutant or combinatorial mutants containing the C26S mutation displayed significantly faster deglutathionylating activity than WT GLRX protein using E-GS-BSA as the substrate (Fig