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Furthermore, transcript levels for parvalbumin were reduced to the same extent in the DLPFC of medication-nave patients compared to patients receiving antipsychotic medication (20)

However, similar symptoms can arise from different aetiological events and pathophysiological mechanisms, causing varied responses to the same therapies

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High glucose suppresses autophagic flux in renal tubular epithelial cells through the mammalian target of rapamycin (mTOR)/Unc-51-like kinase 1 (ULK1) pathway, resulting in the accumulation of undegraded protein aggregates and damaged mitochondria (58)

Regulation of microglia activity by glaucocalyxin-A: attenuation of lipopolysaccharide-stimulated neuroinflammation through NF-B and p38 MAPK signaling pathways