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[DOI] [PubMed] [Google Scholar] Laneuville O
J Mass Spectrom 50:11631174

Braverman O

Weyesa et al
243 During bleomycin-induced pulmonary fibrosis in mice, dysregulated lung microbiota, such as Bacteroides and Prevotella dyregulation, promotes the formation of fibrotic pathogens through IL-17R signaling and drive IL-17B production through their membrane vesicles, thereby promoting lung inflammation and fibrosis

Subsequent studies have found that UA exhibits neuroprotective effects on dopaminergic neurons in experimental PD models by suppressing neuroinflammation and oxidative stress through activation of the Nrf2-ARE antioxidant defense mechanism [176]
