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S.HashemA.E.F

10.1161/circ.148.suppl_1.16908 [DOI] [Google Scholar] 113

In experimental ischemia/reperfusion research, particularly in cases of myocardial infarction and stroke, melatonin has been shown to successfully prevent oxidative damage and the pathophysiological repercussions of such damage are essential (43,82,90,91)

These different routes of administration provide flexibility in how the peptide can be used, making it suitable for a range of medical conditions and patient preferences

However, these models are important because they mimic pathology of NTG, including RGC loss, optic nerve atrophy, and visual impairment with normal IOP 2,3

doi: 10.1002/mnfr.201700854
