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Dr Bortolasci is Alfred Deakin Postdoctoral Research Fellow

Durgapal et al

Given that adult cardiomyocytes still possess a relatively sluggish capacity for cell division, enhancing their ability to proliferate into cardiomyocytes and replace necrotic cardiomyocytes following myocardial ischemia could emerge as a prominent area of focus in future research

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Therefore, we suggest that the reduction of NEFA observed in the glutathione-supplemented mice was due to an increase in muscle utilization rather than a release from adipose tissue