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We hypothesized that high 15LO1, PEBP1, and GPX4 activity drives abnormal asthmatic redox biology, evidenced by lower bronchoalveolar lavage (BAL) fluid and intraepithelial cell GSH:oxidized GSH (GSSG) ratios, to enhance type 2 (T2) inflammatory responses

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Mitochondria related peptide MOTS-c suppresses ovariectomy-induced bone loss via AMPK activation

Mechanistic Studies: Research incorporating neuroimaging and biomarkers is needed to further elucidate the underlying neurobiological mechanisms through which MCE confers its cognitive benefits

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Bethesda, Md.: Cystic Fibrosis Foundation