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Proteomic analysis of colorectal cancer reveals alterations in metabolic pathways: mechanism of tumorigenesis
( B ) Immunoblot analysis PDIA3 and PDIA6 knockdown efficiency for the cells used in a and Fig
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This mechanism promotes the proliferation of ISCs, a process leading to the accumulation of somatic mutations and increasing the risk of benign adenomas progressing to CRC 78
Multiple epigenetic mechanisms are utilized to prevent active transcription of ERVs, including DNA and histone methylation
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