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* Correspondence: Michael C

This anti-CCP production and translocation into the systemic circulation has been proposed to accelerate the development of RA with interstitial lung disease for individuals who are genetically predisposed (e.g., gain-of-function MUC5B promoter variant reducing mucociliary function in small airways responsible for clearing inhaled particles in the lungs (170))

Despite the redundancy accorded to this system by the continued production of JNK2, it appears reasonable to conclude that the JNK1 signaling pathway may be playing a role in T-cell receptor initiated Th cell proliferation, differentiation or apoptosis

doi: 10.1016/j.jsps.2017.04.018, PMID: [DOI] [PMC free article] [PubMed] [Google Scholar] 78.Taofiq O, Rodrigues F, Barros L, Peralta RM, Barreiro MF, Ferreira IC, et al

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