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Stratford and co-workers suggested that HQ is neither a substrate nor an inhibitor of tyrosinase 27 while del Mar Garca-Molina et al
Their coordinated upregulation indicates a transcriptional reprogramming toward enhanced secondary metabolism, potentially contributing to improved oxidative resilience and adaptive stress responses
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Antioxidants , 11 (6), 1121
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Cell polarity-determining proteins Par-3 and PP-1 are involved in epithelial tight junction defects in coeliac disease
