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Additionally, KEAP1 alterations enable cancer cells to survive oxidative stress by activating the NRF2 antioxidant machinery and collaborating with altered KRAS to promote LUAD development [103, 104, 105, 106, 107, 108]

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SNPs on CTH reduce its activity and can limit the pool of cysteine for all pathways, potentially affecting glutathione levels or sulphation

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hydrophila compared to the other pathogenic bacteria (Supplementary Fig

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