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We observed that knockdown of TMLHE decreased the nuclear pool of acetyl-CoA (Fig

Further, DISC1 is not found to be associated with SZ risk in more recent GWAS studies and its relevance to SZ is therefore questionable [131, 139] Other genetic models are conditional knock-outs for neuregulin and its receptor ErbB4, showing distinct SZ-like characteristics

The baseline assessment included a detailed sociodemographic data, medical history, frailty status assessment, physical function, cognitive function, nutritional status, and selected frailty biomarkers