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The source of electrons, in this process, is reduced nicotinamide adenine dinucleotide (NADPH) (36, 38)

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Another important miRNA regulating cell metabolism is miR-378/378*, embedded within gene encoding peroxisome proliferator-activated receptor gamma coactivator 1-beta (PGC-1), a transcriptional regulator of oxidative energy metabolism

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Early therapeutic interventions target the temporal duality of MRIS, aiming to mitigate the initial uncompensated glycolytic acceleration (or glycolytic frenzy) by inhibiting lactate dehydrogenase to prevent acidosis [41]

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