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Glutathione- and apolipoprotein E-grafted liposomes improve drug penetration through the BBB, facilitate the targeting of A-damaged neurons, and reduce P-tau protein expression (Kuo et al., 2021)

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We hypothesize that interventions targeting these metabolic barrierssuch as inhibiting agmatinase (AGMAT) to reduce CAF infiltration as proposed by Wang et al., or selectively depleting glutathione in GPX4-high cloneswill unlock the cytolytic capacity of exhausted T cells [307]