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The parallel decline in endogenous GHK-Cu levels is suggestive, though causation has not been established

Consequently, this could improve cellular function by regulating cellular events, such as cytokine production, cell proliferation, synthesis of DNA and proteins, etc

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By disrupting the interaction between FOXO4 and p53, FOXO4-DRI reactivates apoptosis in aged cells, clearing them from tissues and promoting systemic rejuvenation (1, 2)

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