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Besides, a previous in vitro study (130) showed that chronic taurine treatment also could prevent the development of cavernosal dysfunction after diabetes induction

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2.3 Role of impaired NO signaling and mitochondrial dysfunction in sarcopenia At the molecular level, the reduction in NO signaling constitutes a crucial element that correlates vascular dysfunction with the progression of sarcopenia

Given the interesting findings from mechanistic studies in mouse models, where FMO3 suppression reduced TMAO levels and atherosclerosis [32], further research in larger and more standardized human studies is needed to better understand the genetic regulation of TMAO metabolism and its role in CVD risk

AX may influence Polyunsaturated fatty acid (PUFA) metabolism through the modulation of fatty acid composition and the prevention of oxidative degradation rather than by directly stimulating PUFA biosynthesis [81]