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Cancers Cancer cells are susceptible to perturbation of thiol metabolism, where excessive iron release and subsequent lipid peroxidation contribute to carcinogenesis [224]

*Correspondence: Jan Born [email protected] This article was submitted to Clinical Diabetes, a section of the journal Frontiers in Endocrinology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

The idea that antibiotics can reduce the effects of microbiota and their metabolites on host metabolism via the gutliver axis is the foundation for their use in the treatment of MASLD

Thiolase: The final enzyme in the -oxidation pathway, thiolase, cleaves the 3-ketoacyl-CoA into two molecules of acetyl-CoA

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