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Similar to oral ethanol consumption, endogenous ethanol production has been implicated in the development of non-alcohol-associated fatty liver disease, NAFLD (now known as metabolic dysfunction-associated steatotic liver disease, MASLD), and non-alcohol-associatedsteatohepatitis NASH (now metabolic dysfunction-associated steatohepatitis, MASH) (54, 5863)
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Any disruption of that fold inactivates the hormone
No underlying condition is identified
