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166 Hoffman, N
Regulation of TFAM and nucleoids in the CVDs In myocardial infarction, alteration of physiological functions of mitochondria leads to modification of proteins and lipids and inhibition of energy production, contractile capacity, cell necrosis, or apoptosis
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At-home care can address symptoms, but it can't correct the biomechanical drivers, stimulate tissue regeneration, or reverse structural degeneration that's already occurred
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Positive and negative regulation of T-cell activation through kinases and phosphatases
