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glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

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glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

Glutaminase C has higher catalytic activity than kidney glutaminase and is commonly upregulated in leukemia cells (10, 11)

glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

Mephorbarbital has a tertiary amine and it is converted into phenobarbital which has a secondary amine (Figure 4)

glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

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glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

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glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

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glutathione and acetaminophen toxicity Exposure to all its metabolites upon 10, 15, and 20 mg/kg intravenous acetaminophen in very-preterm infants Increased resistance to acetaminophen hepatotoxicity

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