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Rescue of primary ubiquinone deficiency due to a novel COQ7 defect using 2,4dihydroxybensoic acid

MSH2 in contrast to MLH1 and MSH6 is frequently inactivated by exonic and promoter rearrangements in hereditary nonpolyposis colorectal cancer

For the therapeutic treatment of established atherosclerotic plaques, ApoE/ mice were first fed a high-fat diet for 12 weeks to induce atherosclerosis

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