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Chronic Candida overgrowth progressively depletes secretory IgA (sIgA), the guts primary antibody defence, creating a vicious cycle in which reduced immune surveillance allows further yeast proliferation and biofilm formation

These observations suggest that Cys-SSH and GSSH are produced under physiological conditions, while Cys-SS n H and GSS n H are unstable under such conditions

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[2] [7] Treatment response should be monitored through clinical improvement, reticulocyte count (which typically rises within a week), and full blood count after 8 weeks

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Apoptosis due to inflammation, ERS, and oxidative stress has long been thought to be the main cause of cell dysfunction