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Mutations of R-domain phosphorylation sites lead to loss of cadmium tolerance in Ycf1 11 and to lower open probability (but not overall open duration) and sensitivity to ATP concentration in the Ycf1 homolog CFTR 14,15

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While behavioral, genetic, and environmental factors are also involved, the inhalation of significant amounts of chemical substances, such as nicotine, cotinine, NO, ROS, peroxynitrite, and free radicals originating from organic molecules is likely responsible for most of the systemic damage resulting from smoking

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